Receptor-Interacting Protein Homotypic Interaction Motif-Dependent Control of NF-κB Activation via the DNA-Dependent Activator of IFN Regulatory Factors
William J. Kaiser(Emory University), Edward S. Mocarski(Emory University)
Cited by 272
Related Papers
Toll-like Receptor 3-mediated Necrosis via TRIF, RIP3, and MLKL
|Journal of Biological Chemistry|2013|934
Apoptosis Induced by the Toll-Like Receptor Adaptor TRIF Is Dependent on Its Receptor Interacting Protein Homotypic Interaction Motif
|The Journal of Immunology|2005|365
Proteasome inhibition blocks necroptosis by attenuating death complex aggregation
|Cell Death and Disease|2018|29