A Phospholipase C-γ1–Independent, RasGRP1-ERK–Dependent Pathway Drives Lymphoproliferative Disease in Linker for Activation of T Cells–Y136F Mutant Mice
Robert L. Kortum(Uniformed Services University of the Health Sciences), Connie L. Sommers(Center for Cancer Research)
Cited by 17
Related Papers
miR-155 Controls Lymphoproliferation in LAT Mutant Mice by Restraining T-Cell Apoptosis via SHIP-1/mTOR and PAK1/FOXO3/BIM Pathways
|PLoS ONE|2015|31
Wild-type RAS signaling is an essential therapeutic target in <i>RAS</i> -mutated cancers
|Science Signaling|2025|3
In situ modeling of acquired resistance to RTK/RAS pathway targeted therapies
|bioRxiv (Cold Spring Harbor Laboratory)|2023|2