Loss of BAP1 function leads to EZH2-dependent transformation

Lindsay M. LaFave(Memorial Sloan Kettering Cancer Center), Wendy Béguelin(Cornell University), Richard P. Koche(Memorial Sloan Kettering Cancer Center), Matt Teater(Cornell University), Barbara Spitzer(Memorial Sloan Kettering Cancer Center), Alan Chramiec(Memorial Sloan Kettering Cancer Center), Efthymia Papalexi(Memorial Sloan Kettering Cancer Center), Matthew D. Keller(Memorial Sloan Kettering Cancer Center), Todd Hricik(Memorial Sloan Kettering Cancer Center), Katerina Konstantinoff(Memorial Sloan Kettering Cancer Center), Jean‐Baptiste Micol(Memorial Sloan Kettering Cancer Center), Benjamin H. Durham(Memorial Sloan Kettering Cancer Center), Sarah K. Knutson(Epizyme (United States)), John E. Campbell(Epizyme (United States)), Gil Blum(Memorial Sloan Kettering Cancer Center), Xinxu Shi(Memorial Sloan Kettering Cancer Center), Emma H. Doud(Northwestern University), Andrei V. Krivtsov(Memorial Sloan Kettering Cancer Center), Young Rock Chung(Memorial Sloan Kettering Cancer Center), Inna Khodos(Memorial Sloan Kettering Cancer Center), Elisa de Stanchina(Memorial Sloan Kettering Cancer Center), Ouathek Ouerfelli(Memorial Sloan Kettering Cancer Center), Prasad S. Adusumilli(Memorial Sloan Kettering Cancer Center), Paul M. Thomas(Northwestern University), Neil L. Kelleher(Northwestern University), Minkui Luo(Memorial Sloan Kettering Cancer Center), Heike Keilhack(Epizyme (United States)), Omar Abdel‐Wahab(Memorial Sloan Kettering Cancer Center), Ari Melnick(Cornell University), Scott A Armstrong(Memorial Sloan Kettering Cancer Center), Ross L. Levine(Memorial Sloan Kettering Cancer Center)
Nature Medicine
October 5, 2015
Cited by 398Open Access
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