A primate-specific, brain isoform of KCNH2 affects cortical physiology, cognition, neuronal repolarization and risk of schizophrenia

Stephen J. Huffaker(National Institutes of Health), Jingshan Chen(National Institutes of Health), Kristin K. Nicodemus(National Institutes of Health), Fabio Sambataro(National Institutes of Health), Feng Yang(National Institute of Child Health), Venkata S. Mattay(National Institutes of Health), Barbara K. Lipska(National Institutes of Health), Thomas M. Hyde(National Institutes of Health), Jian Song(National Institutes of Health), Dan Rujescu(Ludwig-Maximilians-Universität München), Ina Giegling(Ludwig-Maximilians-Universität München), Karine R. Mayilyan(Yerevan State Medical University), Morgan J Proust(National Institute of Mental Health), Armen Soghoyan(Yerevan State Medical University), Grazia Caforio(University of Bari Aldo Moro), Joseph H Callicott(National Institute of Mental Health), Alessandro Bertolino(University of Bari Aldo Moro), Andreas Meyer‐Lindenberg(National Institutes of Health), Jay Chang(National Institutes of Health), Yuanyuan Ji(National Institute of Child Health), Michael Egan(National Institute of Mental Health), Terry E. Goldberg(National Institutes of Health), Joel E. Kleinman(National Institutes of Health), Bai Lu(National Institutes of Health), Daniel R. Weinberger(National Institutes of Health)
Nature Medicine
May 1, 2009
Cited by 256Open Access
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