The augmentation of O-GlcNAcylation reduces glyoxal-induced cell injury by attenuating oxidative stress in human retinal microvascular endothelial cells
Guodong Liu(Tongji University), Fang Wang(Shanghai Tenth People's Hospital)
Cited by 25
Related Papers
Long Non-Coding RNA MALAT1 Mediates Transforming Growth Factor Beta1-Induced Epithelial-Mesenchymal Transition of Retinal Pigment Epithelial Cells
|PLoS ONE|2016|83
Oxidative stress‐induced c‐Jun N‐terminal kinase (JNK) activation in tendon cells upregulates MMP1 mRNA and protein expression
|Journal of Orthopaedic Research®|2006|55
O-GlcNAcylation under hypoxic conditions and its effects on the blood-retinal barrier in diabetic retinopathy
|International Journal of Molecular Medicine|2013|34
O-GlcNAcylation Inhibition Upregulates Connexin43 Expression in the Endothelium to Protect the Tight Junction Barrier in Diabetic Retinopathy
|Investigative Ophthalmology & Visual Science|2023|10