Bortezomib Sensitizes Malignant Human Glioma Cells to TRAIL, Mediated by Inhibition of the NF-κB Signaling Pathway
Esther P. Jane(UPMC Hillman Cancer Center), Ian F. Pollack(University of Pittsburgh)
Cited by 75
Related Papers
ABT-737 Synergizes with Bortezomib to Induce Apoptosis, Mediated by Bid Cleavage, Bax Activation, and Mitochondrial Dysfunction in an Akt-Dependent Context in Malignant Human Glioma Cell Lines
|Journal of Pharmacology and Experimental Therapeutics|2012|39
Inhibition of Phosphatidylinositol 3-Kinase/AKT Signaling by NVP-BKM120 Promotes ABT-737–Induced Toxicity in a Caspase-Dependent Manner through Mitochondrial Dysfunction and DNA Damage Response in Established and Primary Cultured Glioblastoma Cells
|Journal of Pharmacology and Experimental Therapeutics|2014|35
Survivin Inhibitor YM-155 Sensitizes Tumor Necrosis Factor– Related Apoptosis-Inducing Ligand-Resistant Glioma Cells to Apoptosis through Mcl-1 Downregulation and by Engaging the Mitochondrial Death Pathway
|Journal of Pharmacology and Experimental Therapeutics|2013|28
Abrogation of Mitogen-Activated Protein Kinase and Akt Signaling by Vandetanib Synergistically Potentiates Histone Deacetylase Inhibitor-Induced Apoptosis in Human Glioma Cells
|Journal of Pharmacology and Experimental Therapeutics|2009|27
Reversing tozasertib resistance in glioma through inhibition of pyruvate dehydrogenase kinases
|Molecular Oncology|2021|23