Identification of the SPLUNC1 ENaC-inhibitory domain yields novel strategies to treat sodium hyperabsorption in cystic fibrosis airway epithelial cultures
Carey Hobbs(University of North Carolina at Chapel Hill), Robert Tarran(University of North Carolina at Chapel Hill)
American Journal of Physiology-Lung Cellular and Molecular Physiology
October 12, 2013
Cited by 82
Related Papers
What are the respiratory effects of e-cigarettes?
|BMJ|2019|503
Identification of BPIFA1/SPLUNC1 as an epithelium-derived smooth muscle relaxing factor
|Nature Communications|2017|50
Cigarette Smoke Exposure Induces Retrograde Trafficking of CFTR to the Endoplasmic Reticulum
|Scientific Reports|2019|33
Vaping-Induced Proteolysis Causes Airway Surface Dehydration
|International Journal of Molecular Sciences|2023|6