Modulation of Alzheimer-Like Synaptic and Cholinergic Deficits in Transgenic Mice by Human Apolipoprotein E Depends on Isoform , Aging, and Overexpression of Amyloid β Peptides But Not on Plaque Formation
Manuel Buttini(Centre Hospitalier de Luxembourg), Lennart Mucke(Gladstone Institutes), Frank M. Longo(Stanford University), Yadong Huang(Jinan University), Margaret Mallory(University of California San Diego), Eliezer Masliah(University of California San Diego), Kristina P Shockley(Gladstone Institutes), Brian W. Jones(Gladstone Institutes), Gui-Qiu Yu(Gladstone Institutes), Tracy T. Yeo(San Francisco VA Medical Center)
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