Tumor Necrosis Factor-α–Converting Enzyme Is a Key Regulator of Agonist-Induced Cardiac Hypertrophy and Fibrosis
Xiang Wang(Huzhou University), Carlos Fernández-Patrón(University of Alberta), Stephan Cooper(L-3 Communications (United States)), Fung L. Chow(L-3 Communications (United States)), Gary D. Lopaschuk(University of Alberta), Zamaneh Kassiri(University of Alberta), Tatsujiro Oka(L-3 Communications (United States)), Jeff Odenbach(University of Alberta)
Cited by 96
Related Papers
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|878
Cardiac fibroblasts, fibrosis and extracellular matrix remodeling in heart disease
|Fibrogenesis & Tissue Repair|2012|806
The cardiac phenotype induced by PPARα overexpression mimics that caused by diabetes mellitus
|Journal of Clinical Investigation|2002|763
Vascular Matrix Metalloproteinase-2 Cleaves Big Endothelin-1 Yielding a Novel Vasoconstrictor
|Circulation Research|1999|364
Assessing Cardiac Metabolism
|Circulation Research|2016|284