cIAP1 and cIAP2 limit macrophage necroptosis by inhibiting Rip1 and Rip3 activation
Scott McComb(Institute for Biological Sciences), Subash Sad(Institute for Biological Sciences)
Cited by 146
Related Papers
Efficient apoptosis requires feedback amplification of upstream apoptotic signals by effector caspase-3 or -7
|Science Advances|2019|268
Inhibition of ROS and upregulation of inflammatory cytokines by FoxO3a promotes survival against Salmonella typhimurium
|Nature Communications|2016|57
Caspase-3 Is Transiently Activated without Cell Death during Early Antigen Driven Expansion of CD8+ T Cells In Vivo
|PLoS ONE|2010|54
Differentiated macrophages acquire a pro-inflammatory and cell death–resistant phenotype due to increasing XIAP and p38-mediated inhibition of RipK1
|Journal of Biological Chemistry|2018|26
Tristetraprolin regulates necroptosis during tonic Toll-like receptor 4 (TLR4) signaling in murine macrophages
|Journal of Biological Chemistry|2020|16