Augmented<b><i>β</i>-Cell Function and Mass in Glucocorticoid-Treated Rodents Are Associated with Increased Islet Ir-<i>β</i></b>/AKT/mTOR and Decreased AMPK/ACC and AS160 Signaling
André Otávio Peres Protzek(Universidade Estadual de Campinas (UNICAMP)), Antônio C. Boschero(Universidade Estadual de Campinas (UNICAMP)), José Maria Costa-Júnior(Université Libre de Bruxelles), Alex Rafacho(Universidade Federal de Santa Catarina), Fernanda Ortis(Universidade de São Paulo), Jean Franciesco Vettorazzi(Universidade Estadual de Campinas (UNICAMP)), Everardo M. Carneiro(Universidade Estadual de Campinas (UNICAMP)), Tiago Gomes Araújo(Universidade Estadual de Campinas (UNICAMP)), Gustavo Jorge dos Santos(Universidade de São Paulo), Luiz Fernando de Rezende(Universidade Estadual de Campinas (UNICAMP))
Cited by 26
Related Papers
The Human Pancreatic Islet Transcriptome: Expression of Candidate Genes for Type 1 Diabetes and the Impact of Pro-Inflammatory Cytokines
|PLoS Genetics|2012|478
Glucocorticoid treatment and endocrine pancreas function: implications for glucose homeostasis, insulin resistance and diabetes
|Journal of Endocrinology|2014|214
A role for the putative cannabinoid receptor GPR55 in the islets of Langerhans
|Journal of Endocrinology|2011|119
Participation of prolactin receptors and phosphatidylinositol 3-kinase and MAP kinase pathways in the increase in pancreatic islet mass and sensitivity to glucose during pregnancy
|Journal of Endocrinology|2004|111
High doses of dexamethasone induce increased β-cell proliferation in pancreatic rat islets
|American Journal of Physiology-Endocrinology and Metabolism|2009|106