Shortage of mitogen-activated protein kinase is responsible for resistance to AP-1 transactivation and transformation in mouse JB6 cells
Chuanshu Huang(Wenzhou Medical University), Zigang Dong(China-US (Henan) Hormel Cancer Institute), Nancy H. Colburn(Gene Therapy Laboratory), Matthew R. Young(NYU Langone Health), Weiya Ma(University of Minnesota)
Cited by 201
Related Papers
MicroRNA-21 (miR-21) post-transcriptionally downregulates tumor suppressor Pdcd4 and stimulates invasion, intravasation and metastasis in colorectal cancer
|Oncogene|2007|1.9k
S6K1- and ßTRCP-Mediated Degradation of PDCD4 Promotes Protein Translation and Cell Growth
|Science|2006|712
MicroRNA-21 promotes cell transformation by targeting the programmed cell death 4 gene
|Oncogene|2008|711
The Transformation Suppressor Pdcd4 Is a Novel Eukaryotic Translation Initiation Factor 4A Binding Protein That Inhibits Translation
|Molecular and Cellular Biology|2002|490
Blocking of tumor promoter-induced AP-1 activity inhibits induced transformation in JB6 mouse epidermal cells.
|Proceedings of the National Academy of Sciences|1994|390