A variant upstream of IFNL3 (IL28B) creating a new interferon gene IFNL4 is associated with impaired clearance of hepatitis C virus

Ludmila Prokunina‐Olsson(National Institutes of Health), Brian Muchmore(National Cancer Institute), Wei Tang(National Cancer Institute), Ruth M. Pfeiffer(National Cancer Institute), Heiyoung Park(National Institutes of Health), Harold Dickensheets(Center for Drug Evaluation and Research), Dianna Hergott(National Cancer Institute), Patricia Porter‐Gill(National Institutes of Health), Adam Mumy(National Cancer Institute), Indu Kohaar(National Cancer Institute), Sabrina Chen(Associated Management Services (United States)), Nathan Brand(National Institutes of Health), McAnthony Tarway(National Institutes of Health), Luyang Liu(National Institutes of Health), Faruk Sheikh(Center for Drug Evaluation and Research), Jacquie Astemborski(Johns Hopkins University), Herbert L. Bonkovsky(Carolinas Medical Center), Brian R. Edlin(National Development and Research Institutes), Charles D. Howell(University of Maryland, Baltimore), Timothy R. Morgan(University of California, Irvine), David L. Thomas(Johns Hopkins University), Barbara Rehermann(National Institutes of Health), Raymond P. Donnelly(United States Food and Drug Administration), Thomas R. OʼBrien(National Cancer Institute)
Nature Genetics
January 6, 2013
Cited by 919Open Access
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