Estrogen Receptor-dependent and Estrogen Receptor-independent Pathways for Tamoxifen and 4-Hydroxytamoxifen-induced Programmed Cell Death
Maria Obrero(University of Illinois Urbana-Champaign), David J. Shapiro(University of Illinois Urbana-Champaign)
Cited by 113
Related Papers
A Functional Serine 118 Phosphorylation Site in Estrogen Receptor-α Is Required for Down-Regulation of Gene Expression by 17β-Estradiol and 4-Hydroxytamoxifen
|Endocrinology|2007|57
In Vitro Genetic Analysis of the RNA Binding Site of Vigilin, a Multi-KH-Domain Protein
|Molecular and Cellular Biology|1998|53
Strong and sustained activation of the anticipatory unfolded protein response induces necrotic cell death
|Cell Death and Differentiation|2018|39
Targeting multidrug-resistant ovarian cancer through estrogen receptor α dependent ATP depletion caused by hyperactivation of the unfolded protein response
|Oncotarget|2016|32
Plasma Membrane Channel TRPM4 Mediates Immunogenic Therapy–Induced Necrosis
|Cancer Research|2023|29