Phosphorylation of RelA/p65 on Serine 536 Defines an IκBα-independent NF-κB PathwayCarl Y. Sasaki(National Institute on Aging), Dan L. Longo(National Institutes of Health)Journal of Biological ChemistryAugust 17, 200510.1074/jbc.m504943200Cited by 329SaveCiteExport RISWatch citationsRelated PapersInvolvement of mTOR in CXCL12 Mediated T Cell Signaling and Migration|PLoS ONE|2011|31Traf1 Induction and Protection from Tumor Necrosis Factor by Nuclear Factor-κB p65 Is Independent of Serine 536 Phosphorylation|Cancer Research|2007|12