Vasculopathy in sickle cell disease: Biology, pathophysiology, genetics, translational medicine, and new research directionsGregory J. Kato(National Institutes of Health), Mark T. Gladwin(Erasmus MC)American Journal of HematologyJune 18, 200910.1002/ajh.21475Cited by 343SaveCiteExport RISWatch citationsRelated PapersLactate dehydrogenase as a biomarker of hemolysis-associated nitric oxide resistance, priapism, leg ulceration, pulmonary hypertension, and death in patients with sickle cell disease|Blood|2005|643Nitric Oxide Scavenging by Red Blood Cell Microparticles and Cell-Free Hemoglobin as a Mechanism for the Red Cell Storage Lesion|Circulation|2011|561