Gene expression signature of c-MYC-immortalized human fibroblasts reveals loss of growth inhibitory response to TGFβ
Myra L. Wang(Cape Town HVTN Immunology Laboratory / Hutchinson Centre Research Institute of South Africa), Carla Grandori(Precision for Medicine (United States)), Kristin Robinson(Fred Hutch Cancer Center), Julja Burchard(Rosetta Stone (United States)), Steven R. Bartz(Rosetta Stone (United States)), Denise A. Galloway(Fred Hutch Cancer Center), Michele A. Cleary(The Kids Research Institute Australia), Ryan Walsh(Texas Neurology)
Cited by 10
Related Papers
The Myc/Max/Mad Network and the Transcriptional Control of Cell Behavior
|Annual Review of Cell and Developmental Biology|2000|1.3k
Personalized <i>In Vitro</i> and <i>In Vivo</i> Cancer Models to Guide Precision Medicine
|Cancer Discovery|2017|964
A Cohort Study of the Risk of Cervical Intraepithelial Neoplasia Grade 2 or 3 in Relation to Papillomavirus Infection
|New England Journal of Medicine|1992|952
Direct activation of TERT transcription by c-MYC
|Nature Genetics|1999|903
Expression analysis with oligonucleotide microarrays reveals that MYC regulates genes involved in growth, cell cycle, signaling, and adhesion
|Proceedings of the National Academy of Sciences|2000|827