Mutant presenilins of Alzheimer's disease increase production of 42-residue amyloid β-protein in both transfected cells and transgenic mice

Martin Citron(Harvard University), David Westaway(University of Toronto), Weiming Xia(Harvard University), George A. Carlson(McLaughlin Research Institute), Thekla S. Diehl(Brigham and Women's Hospital), Georges Lévesque(University of Toronto), Kelly Johnson‐Wood(Athena Group (United States)), Michael Lee(Athena Group (United States)), Peter Seubert(Athena Group (United States)), Angela Davis(McLaughlin Research Institute), Dora Kholodenko(Athena Group (United States)), Ruth Motter(Athena Group (United States)), Robin Sherrington(University of Toronto), Billie J. Perry(McLaughlin Research Institute), Hong Yao(University of Toronto), Robert Strome(University of Toronto), Ivan Lieberburg(Athena Group (United States)), Johanna M. Rommens(University of Toronto), Soyeon Kim(Harvard University), Dale Schenk(Athena Group (United States)), Paul Fraser(University of Toronto), Peter St George‐Hyslop(University of Toronto), Dennis J. Selkoe(Harvard University)
Nature Medicine
January 1, 1997
Cited by 1,265

Abstract


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