iPSC-derived neurons from GBA1-associated Parkinson’s disease patients show autophagic defects and impaired calcium homeostasis

David C. Schöndorf(German Center for Neurodegenerative Diseases), Massimo Aureli(University of Milan), Fiona E. McAllister(Harvard University), Christopher J. Hindley(University of Freiburg), Florian Mayer(University of Tübingen), Benjamin Schmid(German Center for Neurodegenerative Diseases), S. Pablo Sardi, Manuela Valsecchi(University of Milan), Susanna A. Hoffmann(University of Tübingen), Lukas Schwarz(University of Tübingen), Ulrike B. S. Hedrich(University of Tübingen), Daniela Berg(University of Tübingen), Lamya S. Shihabuddin, Jing Hu(University of Tübingen), Jan Pruszak(University of Freiburg), Steven P. Gygi(Harvard University), Sandro Sonnino(University of Milan), Thomas Gasser(Hertie Institute for Clinical Brain Research), Michela Deleidi(University of Tübingen)
Nature Communications
June 6, 2014
10.1038/ncomms5028
Cited by 550Open Access
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