Enhanced expression of 70-kilodalton heat shock protein limits cell division in a sepsis-induced model of acute respiratory distress syndrome*Zohar Bromberg(Yeshiva of Greater Washington), Yoram Weiss(Yeshiva of Greater Washington)Critical Care MedicineJanuary 1, 200810.1097/01.ccm.0000295473.56522.efCited by 36SaveCiteExport RISWatch citationsRelated PapersThe Membrane-Associated Transient Receptor Potential Vanilloid Channel Is the Central Heat Shock Receptor Controlling the Cellular Heat Shock Response in Epithelial Cells|PLoS ONE|2013|59Enhanced Hsp70 Expression Protects against Acute Lung Injury by Modulating Apoptotic Pathways|PLoS ONE|2011|45