JNK Contributes to the Tumorigenic Potential of Human Cholangiocarcinoma Cells through the mTOR Pathway Regulated GRP78 InductionChunhong Feng(Southwest Medical University), Rongyang Dai(North Sichuan Medical University)PLoS ONEFebruary 28, 201410.1371/journal.pone.0090388Cited by 19SaveCiteExport RISWatch citationsRelated PapersPI3K/Akt Promotes GRP78 Accumulation and Inhibits Endoplasmic Reticulum Stress-Induced Apoptosis in HEK293 Cells|Folia Biologica|2010|42Association between the hyperuricemia and nonalcoholic fatty liver disease risk in a Chinese population: A retrospective cohort study|PLoS ONE|2017|41Reduction in activating transcription factor�4 promotes carbon tetrachloride and lipopolysaccharide/D‑galactosamine‑mediated liver injury in mice|Molecular Medicine Reports|2018|5