EYA4 is inactivated biallelically at a high frequency in sporadic lung cancer and is associated with familial lung cancer risk

Iseult Wilson(Occupational Cancer Research Centre), Wan L. Lam(University of British Columbia), Calum MacAulay(BC Cancer Agency), John C. English(University of British Columbia Hospital), Katey S.S. Enfield(The Francis Crick Institute), Ite A. Laird‐Offringa(University of Southern California), Carolyn J. Brown(University of British Columbia), Megan Fuller, William W. Lockwood(University of British Columbia), Daniel T. Starczynowski(Cincinnati Children's Hospital Medical Center), Judit P. Banáth(Occupational Cancer Research Centre), Raj Chari(National Institutes of Health), Adi F. Gazdar(Weatherford College), Suhaida A. Selamat(University of Southern California), Ming You(Houston Methodist), Kevin L. Bennewith(BC Cancer Agency), Kelsie L. Thu, Ming‐Sound Tsao(Kumamoto University), Andrea L. Pusic(Memorial Sloan Kettering Cancer Center), P Liu(Medical College of Wisconsin), Aly Karsan(University of British Columbia), Emily A. Vucic(BC Cancer Agency), Nikolina Radulovich(Ontario Institute for Cancer Research), Ming Zhang(Shihezi University), Timon P.H. Buys(University of British Columbia), John Yee(BC Cancer Agency), Kim M. Lonergan(Occupational Cancer Research Centre), Y A Zhang(Oregon Health & Science University), Michelle A. Anderson(A. Alfred Taubman Health Care Center), David J. Rowbotham(Leicester Royal Infirmary), Stephen Lam(University of British Columbia)
Oncogene
October 7, 2013
Cited by 56


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