Carbon Monoxide Inhibition of Apoptosis during Ischemia-Reperfusion Lung Injury Is Dependent on the p38 Mitogen-activated Protein Kinase Pathway and Involves Caspase 3
Xuchen Zhang(Qingdao University), Patty J. Lee(Icahn School of Medicine at Mount Sinai), Augustine M.K. Choi(Cornell University), Roger J. Davis(University of Massachusetts Chan Medical School), Richard A. Flavell(Yale University), Peiying Shan(Yale University), Jawed Alam(Tulane University), Leo E. Otterbein(Beth Israel Deaconess Medical Center)
Cited by 286
Related Papers
Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome
|Nature Immunology|2010|3k
Autophagy in Human Health and Disease
|New England Journal of Medicine|2013|2.6k
Heme Oxygenase-1/Carbon Monoxide: From Basic Science to Therapeutic Applications
|Physiological Reviews|2006|2.3k
Pro-inflammatory Cytokines and Environmental Stress Cause p38 Mitogen-activated Protein Kinase Activation by Dual Phosphorylation on Tyrosine and Threonine
|Journal of Biological Chemistry|1995|2.2k