Somatic mutations in ATP1A1 and CACNA1D underlie a common subtype of adrenal hypertension

Elena Azizan(University of Cambridge), Hanne Poulsen(Aarhus University), Petronel Tuluc(Universität Innsbruck), Junhua Zhou(University of Cambridge), Michael V. Clausen(Aarhus University), Andreas Lieb(Universität Innsbruck), Carmela Maniero(University of Cambridge), Sumedha Garg(University of Cambridge), Elena G. Bochukova(University of Cambridge), Wanfeng Zhao(National Health Service), Lalarukh Haris Shaikh(University of Cambridge), Cheryl A. Brighton(University of Cambridge), Ada Ee Der Teo(University of Cambridge), Anthony P. Davenport(University of Cambridge), Tanja Dekkers(Radboud University Nijmegen), Bastiaan B.J. Tops(Radboud University Nijmegen), Benno Küsters(Radboud University Nijmegen), Jiří Ceral(Charles University), Giles S.H. Yeo(University of Cambridge), Sudeshna Guha Neogi(National Institute for Health and Care Research), Ian G. McFarlane(National Institute for Health and Care Research), Nitzan Rosenfeld(University of Cambridge), Francesco Marass(University of Cambridge), James Hadfield(University of Cambridge), Wojciech Margas(University College London), Kanchan Chaggar(University College London), Miroslav Solař(Charles University), Jaap Deinum(Radboud University Nijmegen), Annette Dolphin(University College London), I. Sadaf Farooqi(University of Cambridge), Jörg Striessnig(Universität Innsbruck), Poul Nissen(Aarhus University), Morris J. Brown(University of Cambridge)
Nature Genetics
August 4, 2013
Cited by 534Open Access
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