BCR-ABL–induced adhesion defects are tyrosine kinase–independent
Jason A. Wertheim(University of Arizona), Warren S. Pear(University of Pennsylvania)
Cited by 81
Related Papers
The promise of organ and tissue preservation to transform medicine
|Nature Biotechnology|2017|530
Notch Signaling in Cancer
|Cancer Biology & Therapy|2002|341
NOTCH1–RBPJ complexes drive target gene expression through dynamic interactions with superenhancers
|Proceedings of the National Academy of Sciences|2013|266
Pre-TCR signaling inactivates Notch1 transcription by antagonizing E2A
|Genes & Development|2009|192
Long-range enhancer activity determines <i>Myc</i> sensitivity to Notch inhibitors in T cell leukemia
|Proceedings of the National Academy of Sciences|2014|172