c-Abl and Arg induce cathepsin-mediated lysosomal degradation of the NM23-H1 metastasis suppressor in invasive cancer

Leann S. Fiore(University of Kentucky), Rina Plattner(University of Kentucky), J. Robert McCorkle(St. Jude Children's Research Hospital), Sourik S. Ganguly, Dana Richards(University of Kentucky), David M. Kaetzel(University of Kentucky), M L Cibull(University of Kentucky), C.M. Beach(University of Kentucky), James Sledziona(University of Kentucky), Janna M. Neltner(University of Kentucky), C Wang(University of Kentucky)
Oncogene
October 7, 2013
10.1038/onc.2013.399
Cited by 41

Related Papers

c-Abl is activated by growth factors and Src family kinases and has a role in the cellular response to PDGF
|Genes & Development|1999|426
Association of an Inherited Genetic Variant With Vincristine-Related Peripheral Neuropathy in Children With Acute Lymphoblastic Leukemia
|JAMA|2015|285
Activation of Abl Tyrosine Kinases Promotes Invasion of Aggressive Breast Cancer Cells
|Cancer Research|2006|199
NALP3 inflammasome upregulation and CASP1 cleavage of the glucocorticoid receptor cause glucocorticoid resistance in leukemia cells
|Nature Genetics|2015|162
Imatinib Reverses Doxorubicin Resistance by Affecting Activation of STAT3-Dependent NF-κB and HSP27/p38/AKT Pathways and by Inhibiting ABCB1
|PLoS ONE|2013|133