A Novel Interaction between hScrib and PP1γ Downregulates ERK Signaling and Suppresses Oncogene-Induced Cell Transformation
Kazunori Nagasaka(The University of Tokyo), Lawrence Banks(International Centre for Genetic Engineering and Biotechnology)
Cited by 33
Related Papers
Angelman syndrome-associated ubiquitin ligase UBE3A/E6AP mutants interfere with the proteolytic activity of the proteasome
|Cell Death and Disease|2015|48
The high-risk HPV E6 target scribble (hScrib) is required for HPV E6 expression in cervical tumour-derived cell lines
|Papillomavirus Research|2016|27
HPV-16 virions can remain infectious for 2 weeks on senescent cells but require cell cycle re-activation to allow virus entry
|Scientific Reports|2018|9
The TGF-β/UCHL5/Smad2 Axis Contributes to the Pathogenesis of Placenta Accreta
|International Journal of Molecular Sciences|2023|7
Identification and characterisation of novel potential phospho-acceptor sites in HPV-16 E7
|Tumour Virus Research|2023|3