Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells

Manjula Nakka, Nancy L. Weigel(Baylor College of Medicine), Irina U. Agoulnik(University of South Florida)
The International Journal of Biochemistry & Cell Biology
December 24, 2012
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