The primary mechanism of attenuation of bacillus Calmette–Guérin is a loss of secreted lytic function required for invasion of lung interstitial tissue
Tsungda Hsu(Albert Einstein College of Medicine), William R. Jacobs(Albert Einstein College of Medicine), Jeevan Padiyar(Albert Einstein College of Medicine), Celia W. Goulding(Albert Einstein College of Medicine), Suzie Hingley‐Wilson(University of Surrey), Steven C. Derrick(Albert Einstein College of Medicine), Mari Gingery(Albert Einstein College of Medicine), P Morin(Albert Einstein College of Medicine), David Eisenberg(UCLA Health), Mei Chen(First Affiliated Hospital Zhejiang University), Bing Chen(Xinjiang Academy of Agricultural and Reclamation Science), C. Harold King(Albert Einstein College of Medicine), Sheldon L. Morris(Albert Einstein College of Medicine), Carolyn B. Marks(Albert Einstein College of Medicine), Frank M. Collins(Amgen (United States)), Robert G. Russell(Albert Einstein College of Medicine), Annie Z. Dai(Albert Einstein College of Medicine)
Cited by 737
Related Papers
Effects of the <i>obese</i> Gene Product on Body Weight Regulation in <i>ob</i> / <i>ob</i> Mice
|Science|1995|4k
β-Secretase Cleavage of Alzheimer's Amyloid Precursor Protein by the Transmembrane Aspartic Protease BACE
|Science|1999|3.8k
GDNF: a Glial Cell Line-Derived Neurotrophic Factor for Midbrain Dopaminergic Neurons
|Science|1993|3.2k
Functional recovery in parkinsonian monkeys treated with GDNF
|Nature|1996|964
THE ROUTE OF ENTERIC INFECTION IN NORMAL MICE
|The Journal of Experimental Medicine|1974|663