Reciprocal Activation Between PLK1 and Stat3 Contributes to Survival and Proliferation of Esophageal Cancer Cells
Yu Zhang(Center for Food Safety and Applied Nutrition), Ming‐Rong Wang(Capital University of Economics and Business), De‐Chen Lin(Cedars-Sinai Medical Center), Yong–Quan Wang(Chinese Academy of Medical Sciences & Peking Union Medical College), Yanbin Feng(Chinese Academy of Medical Sciences & Peking Union Medical College), Xiaoli Du(Huazhong Agricultural University), Haiyong Peng(University of Florida), Hongbing Zhang(Hebei University of Economics and Business), Tongtong Zhang(South China Agricultural University), Xia Ruan(Wuhan University), Yanqiu Huo(Chinese Academy of Medical Sciences & Peking Union Medical College), Jing–Lu Cui(Chinese Academy of Medical Sciences & Peking Union Medical College), Qimin Zhan(Peking University Cancer Hospital), Chengji Wang(Air Force Medical University)
Cited by 102
Related Papers
The chromatin accessibility landscape of primary human cancers
|Science|2018|1.3k
Loss of Tsc1/Tsc2 activates mTOR and disrupts PI3K-Akt signaling through downregulation of PDGFR
|Journal of Clinical Investigation|2003|479
PDGFRs are critical for PI3K/Akt activation and negatively regulated by mTOR
|Journal of Clinical Investigation|2007|381
TP63, SOX2, and KLF5 Establish a Core Regulatory Circuitry That Controls Epigenetic and Transcription Patterns in Esophageal Squamous Cell Carcinoma Cell Lines
|Gastroenterology|2020|186
ANXA2 promotes esophageal cancer progression by activating MYC-HIF1A-VEGF axis
|Journal of Experimental & Clinical Cancer Research|2018|165