PPARγ/RXRα-Induced and CD36-Mediated Microglial Amyloid-β Phagocytosis Results in Cognitive Improvement in Amyloid Precursor Protein/Presenilin 1 Mice
Mitsugu Yamanaka(University of Bonn), Michael T. Heneka(German Center for Neurodegenerative Diseases)
Cited by 328
Related Papers
Microglia in Alzheimer’s disease
|Journal of Clinical Investigation|2017|937
Nonsteroidal Anti-Inflammatory Drugs and Peroxisome Proliferator-Activated Receptor-γ Agonists Modulate Immunostimulated Processing of Amyloid Precursor Protein through Regulation of β-Secretase
|Journal of Neuroscience|2003|358
The Oral Antidiabetic Pioglitazone Protects from Neurodegeneration and Amyotrophic Lateral Sclerosis-Like Symptoms in Superoxide Dismutase-G93A Transgenic Mice
|Journal of Neuroscience|2005|225
Critical Role of Astroglial Apolipoprotein E and Liver X Receptor-α Expression for Microglial Aβ Phagocytosis
|Journal of Neuroscience|2011|181
<i>NOS2</i>Gene Deficiency Protects from Sepsis-Induced Long-Term Cognitive Deficits
|Journal of Neuroscience|2009|143