Ureteral obstruction in neonatal mice elicits segment-specific tubular cell responses leading to nephron loss11See Editorial by Woolf, p. 761.
François Cachat(University of Virginia), Robert L. Chevalier(University of Virginia), Alice Y. Chang(National Cheng Kung University), Bärbel Lange-Sperandio(LMU Klinikum), Michael S. Forbes(University of Virginia), Barbara A. Thornhill(University of North Carolina at Chapel Hill), Susan C. Kiley(University of Virginia)
Cited by 119
Related Papers
Ureteral obstruction as a model of renal interstitial fibrosis and obstructive nephropathy
|Kidney International|2009|1k
Mechanisms of renal injury and progression of renal disease in congenital obstructive nephropathy
|Pediatric Nephrology|2009|218
Reduced angiotensinogen expression attenuates renal interstitial fibrosis in obstructive nephropathy in mice
|Journal of Clinical Investigation|1999|172
Recovery following relief of unilateral ureteral obstruction in the neonatal rat
|Kidney International|1999|129
Proximal tubular injury and rapid formation of atubular glomeruli in mice with unilateral ureteral obstruction: a new look at an old model
|American Journal of Physiology-Renal Physiology|2011|122