Cyclin D Expression Is Controlled Post-transcriptionally via a Phosphatidylinositol 3-Kinase/Akt-dependent Pathway
Robin C. Muise‐Helmericks(Medical University of South Carolina), Neal Rosen(Memorial Sloan Kettering Cancer Center), Alfonso Bellacosa(Fox Chase Cancer Center), H. Leighton Grimes(Cincinnati Children's Hospital Medical Center), Scott Malstrom(Fox Chase Cancer Center), Philip N. Tsichlis(Molecular Oncology (United States))
Cited by 512
Related Papers
mTOR Inhibition Induces Upstream Receptor Tyrosine Kinase Signaling and Activates Akt
|Cancer Research|2006|2.5k
RAF inhibitors transactivate RAF dimers and ERK signalling in cells with wild-type BRAF
|Nature|2010|1.8k
RAF inhibitor resistance is mediated by dimerization of aberrantly spliced BRAF(V600E)
|Nature|2011|1.4k
BRAF mutation predicts sensitivity to MEK inhibition
|Nature|2005|1.3k
Transduction of interleukin-2 antiapoptotic and proliferative signals via Akt protein kinase
|Proceedings of the National Academy of Sciences|1997|526