Silencing of imprinted CDKN1C gene expression is associated with loss of CpG and histone H3 lysine 9 methylation at DMR-LIT1 in esophageal cancer
Hidenobu Soejima(Saga University), Tsunehiro Mukai(Nishikyushu University), Shiroh Matsukura, Makoto Takeuchi(Osaka International Cancer Institute), Zhao Wei, Takeshi Urano(Shimane University), Yoshihiko Kitajima(Saga University), Keiichiro Joh, Masahiro Nakayama(Osaka International Cancer Institute), Mitsuo Oshimura(Japan Science and Technology Agency), Kohji Miyazaki(Saga University), Tetsuji Nakagawachi(Saga Medical School Hospital), Ken Higashimoto(Saga University)
Cited by 57
Related Papers
An imprinted gene p57KIP2 is mutated in Beckwith–Wiedemann syndrome
|Nature Genetics|1996|385
Genomic imprinting of p57KIP2, a cyclin–dependent kinase inhibitor, in mouse
|Nature Genetics|1995|291
Analysis of germline CDKN1C (p57<sup>KIP2</sup>) mutations in familial and sporadic Beckwith-Wiedemann syndrome (BWS) provides a novel genotype-phenotype correlation
|Journal of Medical Genetics|1999|178
A Comprehensive Analysis of Allelic Methylation Status of CpG Islands on Human Chromosome 21q
|Genome Research|2004|169
Restriction landmark genomic scanning method and its various applications
|Electrophoresis|1993|159