Isoflurane postconditioning improved long-term neurological outcome possibly via inhibiting the mitochondrial permeability transition pore in neonatal rats after brain hypoxia–ischemiaPing Zhao(China Medical University), Zhiyi Zuo(University of Virginia), Meng Ding(China Medical University), Hang Xue(China Medical University), Guofa Ji(China Medical University), Xiaochun Zhao(China Medical University), Weiwei Yu(China Medical University)NeuroscienceSeptember 18, 201410.1016/j.neuroscience.2014.09.006Cited by 33SaveCiteExport RISWatch citationsRelated PapersChronic Intermittent Fasting Improves Cognitive Functions and Brain Structures in Mice|PLoS ONE|2013|144Isoflurane Induces Learning Impairment That Is Mediated by Interleukin 1β in Rodents|PLoS ONE|2012|106NAD+ rescues aging-induced blood-brain barrier damage via the CX43-PARP1 axis|Neuron|2023|95New insights into targeting mitochondria in ischemic injury|APOPTOSIS|2021|86Mitochondria-associated membranes (MAMs): a potential therapeutic target for treating Alzheimer’s disease|Clinical Science|2021|73