Pathophysiological Concentrations of H2O2 Induce NADPH Oxidase 1 (Nox1)- and Apoptosis Signal Regulating Kinase 1 (Ask1)- Dependent Smooth Muscle Hypertrophy
Imad Al Ghouleh(University of Pittsburgh), Patrick J. Pagano(University of Pittsburgh), Loreto Egaña(University of Pittsburgh), Gábor Csányi(Institute of Pharmacology), Giovanna Frazziano(S.P.E.C.I.E.S.), Eric E. Kelley(West Virginia University), Gyun Jee Song(Catholic Kwandong University International St. Mary's Hospital), Andrés Rodríguez(S.P.E.C.I.E.S.), Alessandro Bisello(University of Pittsburgh)
Cited by 0
Related Papers
Nitric oxide directly activates calcium-dependent potassium channels in vascular smooth muscle
|Nature|1994|1.7k
Oxidative stress regulates collagen synthesis and matrix metalloproteinase activity in cardiac fibroblasts
|American Journal of Physiology-Cell Physiology|2001|672
Novel Competitive Inhibitor of NAD(P)H Oxidase Assembly Attenuates Vascular O<sub>2</sub><sup>−</sup>and Systolic Blood Pressure in Mice
|Circulation Research|2001|607
Expression of a Functionally Active gp91phox-Containing Neutrophil-Type NAD(P)H Oxidase in Smooth Muscle Cells From Human Resistance Arteries
|Circulation Research|2002|587
Localization of a constitutively active, phagocyte-like NADPH oxidase in rabbit aortic adventitia: Enhancement by angiotensin II
|Proceedings of the National Academy of Sciences|1997|457