SETD2 loss-of-function promotes renal cancer branched evolution through replication stress and impaired DNA repair
Nnennaya Kanu(The Francis Crick Institute), Charles Swanton(The Francis Crick Institute)
Cited by 178
Related Papers
Sensitive and specific multi-cancer detection and localization using methylation signatures in cell-free DNA
|Annals of Oncology|2020|1.5k
deconstructSigs: delineating mutational processes in single tumors distinguishes DNA repair deficiencies and patterns of carcinoma evolution
|Genome biology|2016|1.3k
Intratumor Heterogeneity: Evolution through Space and Time
|Cancer Research|2012|926
Meta-analysis of tumor- and T cell-intrinsic mechanisms of sensitization to checkpoint inhibition
|Cell|2021|875
Toward understanding and exploiting tumor heterogeneity
|Nature Medicine|2015|770