Learning Decreases Aβ*56 and Tau Pathology and Ameliorates Behavioral Decline in 3xTg-AD Mice
Lauren M. Billings(University of California, Irvine), Frank M. LaFerla(University of California, Irvine)
Cited by 138
Related Papers
Lipopolysaccharide-Induced Inflammation Exacerbates Tau Pathology by a Cyclin-Dependent Kinase 5-Mediated Pathway in a Transgenic Model of Alzheimer's Disease
|Journal of Neuroscience|2005|686
Nicotinamide Restores Cognition in Alzheimer's Disease Transgenic Mice via a Mechanism Involving Sirtuin Inhibition and Selective Reduction of Thr231-Phosphotau
|Journal of Neuroscience|2008|401
Temporal Profile of Amyloid-β (Aβ) Oligomerization in an in Vivo Model of Alzheimer Disease
|Journal of Biological Chemistry|2005|376
Dietary Docosahexaenoic Acid and Docosapentaenoic Acid Ameliorate Amyloid-β and Tau Pathology via a Mechanism Involving Presenilin 1 Levels
|Journal of Neuroscience|2007|318
SERCA pump activity is physiologically regulated by presenilin and regulates amyloid β production
|The Journal of Cell Biology|2008|300