Skeletal muscle-specific overproduction of constitutively activated c-Jun N-terminal kinase (JNK) induces insulin resistance in mice
Darren C. Henstridge(University of Tasmania), Mark A. Febbraio(Molecular Research Institute)
Cited by 65
Related Papers
Interleukin-6 Increases Insulin-Stimulated Glucose Disposal in Humans and Glucose Uptake and Fatty Acid Oxidation In Vitro via AMP-Activated Protein Kinase
|Diabetes|2006|880
Interleukin-6 Stimulates Lipolysis and Fat Oxidation in Humans
|The Journal of Clinical Endocrinology & Metabolism|2003|758
Brain-derived neurotrophic factor is produced by skeletal muscle cells in response to contraction and enhances fat oxidation via activation of AMP-activated protein kinase
|Diabetologia|2009|708
Extracellular Vesicles Provide a Means for Tissue Crosstalk during Exercise
|Cell Metabolism|2018|648
Searching for the exercise factor: is IL-6 a candidate?
|Journal of Muscle Research and Cell Motility|2003|552