β Subunits of Voltage-gated Sodium Channels Are Novel Substrates of β-Site Amyloid Precursor Protein-cleaving Enzyme (BACE1) and γ-Secretase
Hon Kit Wong(Harvard University), Nobuyuki Nukina(Juntendo University)
Cited by 292
Related Papers
Complete Loss of Post-translational Modifications Triggers Fibrillar Aggregation of SOD1 in the Familial Form of Amyotrophic Lateral Sclerosis
|Journal of Biological Chemistry|2008|212
The pathogenic mechanisms of polyglutamine diseases and current therapeutic strategies
|Journal of Neurochemistry|2009|177
Anti–Vascular Endothelial Growth Factor Therapies as a Novel Therapeutic Approach to Treating Neurofibromatosis-Related Tumors
|Cancer Research|2010|117