Classic and atypical fibrodysplasia ossificans progressiva (FOP) phenotypes are caused by mutations in the bone morphogenetic protein (BMP) type I receptor ACVR1
Frederick S. Kaplan(University of Pennsylvania), Eileen M. Shore(Fisher & Paykel Healthcare (New Zealand)), Julie Hoover‐Fong(Johns Hopkins University), Stefan Mundlos(Max Planck Institute for Molecular Genetics), William Reardon(Our Lady's Hospital), Michael Zasloff(University of Pennsylvania), Stephen J. Forman(City Of Hope National Medical Center), Petra Seemann(Max Planck Institute for Molecular Genetics), Bernhard Köster(Klinikum Lüdenscheid), Rolf Morhart(Garmisch-Partenkirchen Medical Center), Patricia Delai(Irmandade da Santa Casa de Misericórdia de São Paulo), Richard M. Pauli(University of Wisconsin–Madison), Jay C. Groppe(Gaston College), Liam Carroll(Nenagh Hospital), Meiqi Xu(University of Pennsylvania), Syed-Adeel Zaidi(Indiana University – Purdue University Indianapolis), Gabriele Gillessen‐Kaesbach(University Hospital Schleswig-Holstein), Elisabeth Fastnacht-Urban(Klinikum Lüdenscheid), J. M. Connor(University of Glasgow), David L. Glaser(University of Pennsylvania)
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