Substrates modified by advanced glycation end-products cause dysfunction and death in retinal pericytes by reducing survival signals mediated by platelet-derived growth factor
Alan W. Stitt(Royal Victoria Hospital), Tom A. Gardiner(University of Ulster)
Cited by 61
Related Papers
Diabetic retinopathy: current understanding, mechanisms, and treatment strategies
|JCI Insight|2017|1k
Hyperglycaemia-induced pro-inflammatory responses by retinal Müller glia are regulated by the receptor for advanced glycation end-products (RAGE)
|Diabetologia|2010|199
Myeloid Angiogenic Cells Act as Alternative M2 Macrophages and Modulate Angiogenesis through Interleukin-8
|Molecular Medicine|2011|181