Advanced Glycation End Products Increase Permeability of Brain Microvascular Endothelial Cells through Reactive Oxygen Species–Induced Vascular Endothelial Growth Factor Expression
Yoshimasa Niiya(Hokkaido University), Masayoshi Takeuchi(Kanazawa Medical University), Sho‐ichi Yamagishi(Kurume University), Takeo Abumiya(Hokkaido University), Jun-ichi Takino(Hiroshima International University)
Journal of Stroke and Cerebrovascular Diseases
February 6, 2011
Cited by 37
Related Papers
Matrix Metalloproteinases Increase Very Early during Experimental Focal Cerebral Ischemia
|Journal of Cerebral Blood Flow & Metabolism|1999|552
Suppression of transforming growth factor beta and vascular endothelial growth factor in diabetic nephropathy in rats by a novel advanced glycation end product inhibitor, OPB-9195
|Diabetologia|1999|214
Atorvastatin decreases serum levels of advanced glycation endproducts (AGEs) in nonalcoholic steatohepatitis (NASH) patients with dyslipidemia: clinical usefulness of AGEs as a biomarker for the attenuation of NASH
|Journal of Gastroenterology|2010|160
Activated Microvessels Express Vascular Endothelial Growth Factor and Integrin α <sub>v</sub> β <sub>3</sub> During Focal Cerebral Ischemia
|Journal of Cerebral Blood Flow & Metabolism|1999|155
Rapid Loss of Microvascular Integrin Expression during Focal Brain Ischemia Reflects Neuron Injury
|Journal of Cerebral Blood Flow & Metabolism|2001|141