Sole BCR-ABL inhibition is insufficient to eliminate all myeloproliferative disorder cell populations
Sook S. Wong(Howard Hughes Medical Institute), Owen N. Witte(Howard Hughes Medical Institute), Cuilin Zhang(National University of Singapore), Kevan M. Shokat(Howard Hughes Medical Institute), D. Cheng(Howard Hughes Medical Institute), J McLaughlin(Howard Hughes Medical Institute)
Cited by 71
Related Papers
K-Ras(G12C) inhibitors allosterically control GTP affinity and effector interactions
|Nature|2013|2.5k
RAF inhibitors transactivate RAF dimers and ERK signalling in cells with wild-type BRAF
|Nature|2010|1.8k
IRE1 Signaling Affects Cell Fate During the Unfolded Protein Response
|Science|2007|1.4k