The cell biology of β-amyloid precursor protein and presenilin in Alzheimer's diseaseDennis J. Selkoe(Brigham and Women's Hospital)Trends in Cell BiologyNovember 1, 199810.1016/s0962-8924(98)01363-4Cited by 857SaveCiteExport RISWatch citationsAbstractRelated PapersAmyloid β-peptide is produced by cultured cells during normal metabolismChristian Haass, Michael G. Schlossmacher, Albert Y. Hung et al.|Nature|1992|1.9kDeficiency of presenilin-1 inhibits the normal cleavage of amyloid precursor proteinBart De Strooper, Paul Säftig, Katleen Craessaerts et al.|Nature|1998|1.8kVisualization of Aβ42(43) and Aβ40 in senile plaques with end-specific Aβ monoclonals: Evidence that an initially deposited species is Aβ42(43)Takeshi Iwatsubo, Asano Odaka, Nobuhiro Suzuki et al.|Neuron|1994|1.8kAlzheimer's disease and Down's syndrome: Sharing of a unique cerebrovascular amyloid fibril proteinGeorge G. Glenner, Caine W. Wong|Biochemical and Biophysical Research Communications|1984|1.7kNotch-1 signalling requires ligand-induced proteolytic release of intracellular domainEric H. Schroeter, Jeffrey A. Kisslinger, Raphael Kopan|Nature|1998|1.6k