The PSD protein ProSAP2/Shank3 displays synapto-nuclear shuttling which is deregulated in a schizophrenia-associated mutation
Stefanie Grabrucker(University College Cork), Andreas M. Grabrucker(University of Limerick), Tobias M. Boeckers(University of Münster), Matti Eckert(Universität Ulm), Michael J. Schmeißer(Leibniz Institute for Neurobiology), Christian Proepper(Universität Ulm), Katharina Mangus(Universität Ulm), Resham Chhabra(Universität Ulm)
Cited by 65
Related Papers
Haploinsufficiency of TBK1 causes familial ALS and fronto-temporal dementia
|Nature Neuroscience|2015|782
Genetic and Functional Analyses of SHANK2 Mutations Suggest a Multiple Hit Model of Autism Spectrum Disorders
|PLoS Genetics|2012|443
ProSAP/Shank proteins – a family of higher order organizing molecules of the postsynaptic density with an emerging role in human neurological disease
|Journal of Neurochemistry|2002|348