Increased atherosclerosis in mice reconstituted with apolipoprotein E null macrophages
Sergio Fazio(Alzheimer's Association), MacRae F. Linton(Vanderbilt University), Alisa B. Murray(Vanderbilt University), Vladimir R. Babaev(Vanderbilt University Medical Center), Alyssa H. Hasty(Vanderbilt University), Linda A. Gleaves(Pulmonary and Allergy Associates), James B. Atkinson(AstraZeneca (United Kingdom)), Kathy J. Carter(Vanderbilt University)
Cited by 289
Related Papers
Current Perspectives on Statins
|Circulation|2000|1.2k
Treatment of diabetes and atherosclerosis by inhibiting fatty-acid-binding protein aP2
|Nature|2007|732
Lack of macrophage fatty-acid–binding protein aP2 protects mice deficient in apolipoprotein E against atherosclerosis
|Nature Medicine|2001|692
Reducing endoplasmic reticulum stress through a macrophage lipid chaperone alleviates atherosclerosis
|Nature Medicine|2009|482
Repetitive intratracheal bleomycin models several features of idiopathic pulmonary fibrosis
|American Journal of Physiology-Lung Cellular and Molecular Physiology|2010|311