Structural interpretation of the mutations in the beta-cardiac myosin that have been implicated in familial hypertrophic cardiomyopathy.

Ivan Rayment(University of Wisconsin–Madison), Nava Epstein(Sheba Medical Center), James R. Sellers(National Heart Lung and Blood Institute), Lameh Fananapazir(National Heart Lung and Blood Institute), Hazel M. Holden(University of Wisconsin–Madison)
Proceedings of the National Academy of Sciences
April 25, 1995
10.1073/pnas.92.9.3864
Cited by 225

Related Papers

Mechanism of Blebbistatin Inhibition of Myosin II
|Journal of Biological Chemistry|2004|1k
Targeted integration of adeno‐associated virus (AAV) into human chromosome 19.
|The EMBO Journal|1991|774
Long-term results of dual-chamber (DDD) pacing in obstructive hypertrophic cardiomyopathy. Evidence for progressive symptomatic and hemodynamic improvement and reduction of left ventricular hypertrophy.
|Circulation|1994|383
Channeling of Substrates and Intermediates in Enzyme-Catalyzed Reactions
|Annual Review of Biochemistry|2001|379
The Enolase Superfamily:  A General Strategy for Enzyme-Catalyzed Abstraction of the α-Protons of Carboxylic Acids
|Biochemistry|1996|307