Endogenous IL-11 Signaling Is Essential in Th2- and IL-13–Induced Inflammation and Mucus Production
Chun Geun Lee(Yale University), Jack A. Elias(Brown University), Dominik Hartl(Yale University), Robert Homer(Yale University), Felicity Dunlop(Deakin University), Pierre Scotney, Ning‐Yuan Chen(Yale University), Hiroshi Matsuura(Yale University), Qingsheng Chen(Yale University), Chuyan Tang(Yale University), Manuel Baca(Tri-Institutional Therapeutics Discovery Institute), Andrew D. Nash(Walter and Eliza Hall Institute of Medical Research), Louis Fabri(Walter and Eliza Hall Institute of Medical Research)
Cited by 75
Related Papers
Pulmonary expression of interleukin-13 causes inflammation, mucus hypersecretion, subepithelial fibrosis, physiologic abnormalities, and eotaxin production
|Journal of Clinical Investigation|1999|1.7k
Interleukin-13 Induces Tissue Fibrosis by Selectively Stimulating and Activating Transforming Growth Factor β1
|The Journal of Experimental Medicine|2001|926
Airway remodeling in asthma
|Journal of Clinical Investigation|1999|703
Inducible targeting of IL-13 to the adult lung causes matrix metalloproteinase– and cathepsin-dependent emphysema
|Journal of Clinical Investigation|2000|665
Neutrophils: Between Host Defence, Immune Modulation, and Tissue Injury
|PLoS Pathogens|2015|658