IFN-α and Bortezomib Overcome Bcl-2 and Mcl-1 Overexpression in Melanoma Cells by Stimulating the Extrinsic Pathway of Apoptosis

Gregory B. Lesinski(The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute), William E. Carson(The Ohio State University Comprehensive Cancer Center – Arthur G. James Cancer Hospital and Richard J. Solove Research Institute)
Cancer Research
October 15, 2008
10.1158/0008-5472.can-08-0426
Cited by 53

Related Papers

IL-6 and PD-L1 antibody blockade combination therapy reduces tumour progression in murine models of pancreatic cancer
|Gut|2016|540
Psychological, Behavioral, and Immune Changes After a Psychological Intervention: A Clinical Trial
|Journal of Clinical Oncology|2004|415
Generation of monocyte-derived tumor-associated macrophages using tumor-conditioned media provides a novel method to study tumor-associated macrophages in vitro
|Journal for ImmunoTherapy of Cancer|2019|181
The antitumor effects of IFN-α are abrogated in a STAT1-deficient mouse
|Journal of Clinical Investigation|2003|140
The small molecule curcumin analog FLLL32 induces apoptosis in melanoma cells via STAT3 inhibition and retains the cellular response to cytokines with anti-tumor activity
|Molecular Cancer|2010|115